Most patients have symptoms prior to an actual heart attack - generally a "tightness" behind the breastbone, which generally is caused by an increase in heart rate. The coronary arteries fill in between heart beats therefore whatever which increases your heart rate will cut the time your coronary arteries have to furnish blood flow to the heart muscle- the myocardium. Once the heart muscle is deprived of oxygen it becomes irritable foremost to palpitations and, if not relieved, death of the muscle.
Surrounding the dead muscle is a variable sized area of heart muscle at risk of dying because of inadequate oxygen furnish - this condition is known as myocardial ischaemia.
The heart rate is under the control of the autonomic nervous systems, which, in the case of the heart, is driven by adrenaline the so-called "flight or fight" hormone.
Adrenaline acts at assorted sites of the body
- On the sinus node in the heart which controls rate to increase heart rate
- On the bronchial tubes in the lung to cause them to dilate allowing greater flow of air into the lungs
- On the small arteries in the skin and gut to constrict (narrow) reducing blood flow to these areas and development more blood available to the muscles- to fight or fly
- Increases the blood sugar by increasing breakdown of glycogen to glucose, foremost food for the muscles to ageement better.
Whilst increased adrenaline output is tasteless with exercise it also occurs in states of shock, fear, or anger producing gooseflesh and sweating (remember the first time you saw the shower scene in the Hitchcock Movie Psycho).
As well as increasing the heart rate adrenaline may cause cardiac irregularity, increased blood pressure, or palpitations. It is this irregularity which may lead the heart to chaotic beats and finally fibrillation and death.
This is the first indication of illness of a heart attack in about 20% of habitancy - sudden death - the victim just collapses to the ground. Whilst he may gasp a few breaths he has no cardiac output- no blood is leaving his heart- he is pulseless.
This concluding condition (ventricular fibrillation) is very undoubtedly treated by first aid measures and cardiac shock- termed defibrillation. Nowadays many areas which have high populations (or as in aeroplanes, confined areas) have habitancy kindly defibrillators available which only shock patients who have rhythms which are treatable by shock. These machines have easy instructions and any bystander who can read can apply the electrodes and save the patient's life.
Often habitancy wrongly refer to this cause of death as a gigantic heart attack- it takes some time (often up to an hour) to die from a gigantic heart attack. In this case the heart attack causes such valuable damage to the main ventricle of the heart as to cut blood pressure to critically low levels and death results.
Cause of Heart Attacks
The pathological cause of heart attacks is a clot of blood, which forms over an area on the lining of a coronary artery which, is "ready" for a clot to form - this area is termed a vulnerable plaque.
It was previously idea that clots advanced on the lining of arterial walls that had become calcified and thickened - it is now known that rarely are these areas the sites of clots.
Calcification and deposition of cholesterol within the arterial wall reduces the lumen size and therefore flow of blood straight through this section of the artery is likewise reduced. This reduced flow may be sufficient to furnish sufficient oxygen to the heart muscle during rest or light work. Any way once the heart is required to work harder the blood flow is insufficient to furnish sufficient oxygen to the heart muscle and symptoms follow.
Angina- Pectoris and Decubitus
When the heart muscle receives insufficient oxygen it produces lactic and pyruvic acids and these produces the tightness in the chest known as angina the type of angina which occurs during exercise is termed Angina Pectoris.
The definite rehabilitation is to cut the heart work by ceasing whatever performance has caused the heart rate to rise.
Whilst typically angina pectoris occurs as tightness in the chest it may move up into the throat or down one or both arms. Occasionally it may only occur in the teeth or in the back between the shoulder blades. Patients may report it as a "weight on my chest" or "constriction". Occasionally the only indication of illness may be acute breathlessness.
The cardinal highlight is the relation to increased heart work and its relief by stopping activity. In increasing one should use a trintrin preparation. If pain is gift use someone else dose after five minutes or so and repeat twice.
The rehabilitation of angina is beyond the scope of this description Any way drugs which cut the work the heart has to do (like putting a governor on your heart) are most efficient and as they block the performance of adrenaline are termed betablockers.
Drugs, which dilate the coronary arteries (trintrins), are also helpful. These come as patches, tablets for sucking, sprays for inhalation or as solutions for intravenous use in hospital. These should be used only when the tightness is gift and not when you feel giddy or weak as they will cut blood pressure and may cause you to faint. These also dilate all arteries and therefore may cause headache.
Trintrins are particularly risky when used in conjunction with some of the newer drugs for erectile dysfunction eg viagra and these must not be used within 24 hours of each other
Spasm of the coronary arteries may also cause discount in blood flow to the heart. Every person has some degree of spasm within their bodies and angina is generally due to composition of both atheroma and spasm.
However angina, which occurs at rest particularly at night colse to 2.0am waking the patient, is due to vasospasm and is termed angina decubitus.
The symptoms are similar to angina pectoris but in this case it is spasm of the artery and the rehabilitation is not betablockade therapy but a group of drugs called calcium antagonists. These drugs ease the spasm of the artery.
Unstable Angina
Should the pain be continued and not relieved by rest and trintrin therapy the term unstable angina is used.
In this case the pain is more severe and often linked with an increase in sympathetic overactivity. Symptoms such as sweating, nausea, vomiting, palpitations, faintness or a feeling of wanting to faint (presyncope), or even syncope are common.
This is a medical accident and an ambulance should be called without delay, as it is impossible to differentiate unstable angina from a heart attack without electrocardiograph and blood studies.
If you believe that you may have this syndrome whilst waiting for the ambulance chew a whole aspirin tablet.
This condition is generally linked with a clot being formed within one of the main coronary arteries and hospitalisation is valuable so that a definitive pathology may be made and urgent rehabilitation to safe the heart from damage instituted.
Differential Diagnosis
Angina is often confused with indigestion and many patients rationalise that whilst they may reconsider the pain could be angina it "simply cannot happen to me' and it is probably only indigestion or just wind.
One eminent Professor of Cardiology writing in one of the journals described how he waited for eighteen hours before going to Hospital as he idea it " was plainly bad indigestion". He accomplished up with bypass surgery!
Indigestion is generally in the pit of the stomach burning in nature, often relieved by belching or by drinking a glass of milk or alkali. It has no relation to exercise, rarely referred to the arms or back, and may come up into throat as a burning sensation. Trintrins have no effect.
Hiatus hernia or oesophageal reflux may mimic vasospastic angina as they both occur at night when the sick person is prone.
Silent Ischaemia
Some patients, generally those with high pain thresholds, may have myocardial ischaemia which is entirely asymptomatic.
Rarely patients may be found with classical electrocardiographic changes of a heart attack without remembering whatever remotely like the symptoms, which are noted above.
On a treadmill exercise test these patients may manufacture changes on their cardiograph, which are diagnostic of myocardial ischaemia. They may manufacture high blood pressure or more dangerous, a fall in blood pressure during exercise without any actual pain.
These patients are very difficult to treat, as, unlike most of us, they do not get chest tightness of pressure before they manufacture risky cardiac abnormalities. These habitancy wish invasive or nuclear studies to ascertain the workload that causes trouble. They can then be warned to control their work level.
Heart Failure and Cardiac Irregularities
Patients in their senior years may manufacture heart failure without any pain or tightness. The Electrocardiographic changes of a old heart attack are found on disposition examination.
Atrial fibrillation, a not uncommon rhythm disturbance, often has myocardial ischaemia as its basic cause, particularly in the elderly. This irregularity results in a discount in the output from the left ventricle (stroke volume). As blood continues to fill the ventricle from above the lower output results in the heart becoming distended with blood.
Heart muscle like all other muscles when stretched contracts with greater impel thereby increasing the output. The sick person is unaware of whatever the matter with his heart until an xray is taken showing an enlarged heart . (Compensated Heart Failure)
However if the guess for the discount in stroke volume remains then finally (when the heart cannot enlarge further) pressure within the ventricle rises and is passed backwards straight through the atrium and on into the pulmonary arterioles causing fluid to move into the air sacs of the lungs and the patients notices increasing shortness of breath breathlessness. (Decompensated Heart Failure)
Summary
- Never take chest pain lightly - you might wake up dead!
- Learn a plan and keep to it.
- It is only at an accident Centre that you can get immediate diagnosis.
- Most Heart Attacks who are destined to die do so within the first few hours. Don't Delay.
- special rehabilitation to cut the residual damage to your heart is most efficient when given early. Particularly within three hours of the onset of pain.
- You will not be laughed at for going to the Hospital with indigestion.
- Remember no one can exclude a Heart attack from a Severe anginal pain without Ecg and blood tests.
Hiatus Hernia Surgery:How Do I Know If I Am Having a Heart Attack?
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